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Mice heterozygous for mitochondrial trifunctional protein have compromised mitochondrial -oxidation and develop hepatic steatosis alongside a compensatory upregulation of CYP2E1-facilitated FAO and oxidative stress [101, 102]

This manufacturing capacity build-out is a critical enabling factor for the projected market growth trajectory through 2034, as insufficient supply has historically been one of the primary constraints on semaglutide market penetration

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